Researchers found that hawthorn could reduce fatty liver, the levels of triglyceride, cholesterol, and glucose, and oxidative stress damage in animal models. This study demonstrates the potential of hawthorn as a therapeutic agent in the treatment of hyperlipidemia (excess of lipids, such as cholesterol and triglycerides, in the blood).
研究人员发现，山楂可减少动物模型的脂肪肝现象，并降低三酸甘油酯、胆固醇以及葡萄糖的水平，同时减轻氧化压力造成的损伤。这项研究显示了山楂有潜力成为高脂血症（血液中过多的脂质，例如胆固醇和三酸甘油酯）的治疗剂。
September 2022
2022年9月
https://doi.org/10.3389/fnut.2022.936229
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原花青素（OPCs）
原花青素（OPCs）具有很強的清除自由基的能力。它们有可能在体内进化，并且变成对人体有害的细菌。研究显示，某些益生菌会攻击肠道保护膜，并增加罹患肠躁症候群等疾病的风具有很强的清除自由基的能力。自由基会破坏人体细胞，甚至会对我们的遗传物质DNA造成损害。原花青素可以保护肝脏免受有毒物质的伤害，并保护肝脏免于脂肪肝等疾病的侵袭。其它类型的植物性营养，如：黄烷醇、黄酮醇和异黄酮等黄酮类的物质，以及非黄酮类的物质，如：没食子酸，也可以预防包括肝癌在内的各类肝脏疾病。这些植物性营养在多种食物中都可以找到，例如：山楂、枳椇子、草莓、葡萄和大豆等。
Therapeutic Potential of Natural Antioxidants
Oligomeric proanthocyanidins (OPCs) have strong free radical scavenging abilities. Free radicals can damage our body’s cells, and even our DNA. OPCs can protect the liver against injuries from toxic substances, and also protect it from fatty liver disease. Other plant nutrients like flavonoids, such as flavanonols, flavonols, and isoflavones, and nonflavonoids like gallic acid, may also protect against liver diseases, including liver cancer. These plant nutrients can be found in a variety of foods, such as hawthorn, Hovenia dulcis, strawberries, grapes, and soy.
Note:
Liver disease, involving a wide range of liver pathologies from fatty liver, hepatitis, and fibrosis to cirrhosis and hepatocellular carcinoma, is a serious health problem worldwide. In recent years, many natural foods and herbs with abundant phytochemicals have been proposed as health supplementation for patients with hepatic disorders.
Alcoholic liver disease (ALD) is one of the most important causes of liver-related death. In one the study, Oligomeric proanthocyanidins, a set of bioflavonoid complexes having strong free radical scavenging ability, protected liver from alcohol-induced injury and steatosis through decreasing the expressions of lipid synthesis genes and inflammation genes.
Source:
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